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In one study, it has been shown that in an acidic environment, macrophages exposed to bacterial lipopolysaccharides release more cytokines, such as tumor necrosis factor (TNFα) or interferon-γ (IFN-γ) leads to increased expression of the inducible isoform of nitric oxide synthase (iNOS or NOS II), which is responsible for the increased production of nitric oxide (NO) [72,73]
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The post adds that GLP-1 therapy may raise serum uric acid through weight-loss-related shifts in purine metabolism
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