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) applied at the beginning of reperfusion (2 h ischemia/2 h reperfusion) decreased infarct area, 3-nytrotirosyne levels, and prevented the decrease in antioxidant enzymes (glutathione peroxidase and superoxide dismutase) [81]
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340 Metabolic profiling shows that TGF-β1-activated fibroblasts display enhanced glycolysis and glutaminolysis with concurrent succinate accumulation, correlating with a profibrotic phenotype, while itaconate treatment significantly suppresses collagen synthesis, suggesting antagonism of the TGF-β1-glycolysis/glutaminolysis-succinate axis
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